{"id":264,"date":"2020-05-26T19:55:21","date_gmt":"2020-05-26T19:55:21","guid":{"rendered":"https:\/\/carleton.ca\/neuroscience\/?post_type=cu_people&p=264"},"modified":"2025-09-22T11:48:46","modified_gmt":"2025-09-22T15:48:46","slug":"hongyu-sun","status":"publish","type":"cu_people","link":"https:\/\/carleton.ca\/neuroscience\/people\/hongyu-sun\/","title":{"rendered":"Hongyu Sun"},"content":{"rendered":"
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Dr. Hongyu Sun received his MD from Tianjin University in 1997 and his PhD in Neuroscience from Carleton University in 2008. He completed his postdoctoral training at Boston Children’s Hospital \/ Harvard Medical School. He later joined the Carleton Neuroscience department in 2016.<\/p>\n\n\n\n

Eligible to supervise <\/strong>at the undergraduate and graduate level.<\/p>\n\n\n\n

Courses Taught:<\/h3>\n\n\n\n
  • NEUR 2002: Introduction to Statistics in Neuroscience<\/li><\/ul>\n\n\n\n

    Scholarly Work & Research Interests:<\/h3>\n\n\n\n

    The Sun Lab focuses on understanding how early-life experiences affect brain development and cause long-term psychiatric and neurological consequences. They use a combination of electrophysiological, behavioral, neuroimaging, modeling, and molecular biology techniques to address these questions.<\/p>\n\n\n\n

    • A predominant focus of the Sun Lab is early-life epilepsy
      • Investigating whether a selective neuronal ensemble mediates the pathophysiology of early life epilepsy<\/li>
      • Discovering novel molecular signaling pathways<\/li><\/ul><\/li>
      • Interaction between early-life stress and brain development
        • Examining how early-life stress during the critical period reshapes the development of glutamatergic synapses and causes long-term cognitive deficits<\/li><\/ul><\/li><\/ul>\n\n\n\n

          Selected publications:<\/h3>\n\n\n\n

          Sun H, Takesian A, Wang TT, Lippman-Bell JJ, Hensch TK, Jensen FE. (2018) Early seizures prematurely unsilence auditory synapses to disrupt thalamocortical critical period plasticity. Cell Report<\/em>. 23(9):2533-2540.<\/p>\n\n\n\n

          Sun H, Juul H, Jensen FE. (2016) Models of hypoxia and ischemia-induced seizures. Journal of Neuroscience Method<\/em>, 15;260:252-60.<\/p>\n\n\n\n

          Sun H, Kosaras B, Klein PM, Jensen FE. (2013). Mammalian target of rapamycin complex 1 activation negatively regulates Polo-like kinase 2-mediated homeostatic compensation following neonatal seizures. PNAS<\/em>, 110(13): 5199-204.<\/p>\n\n\n\n

          Talos DM, Sun H<\/em>, Zhou X*, Fitzgerald E, Jackson M, Lan V, Joseph A, Jensen FE. (2012) Inhibition of the mammalian target of rapamycin (mTORC1) pathway prevents later life epilepsy and autism-like behavior following early life seizures. PLoS ONE<\/em>, 7(5): e35885. doi:10.1371\/journal.pone.0035885.<\/p>\n\n\n\n

          Zhou C, Lippman Bell JJ<\/em>, Sun H*, Jensen FE. (2011) Hypoxia-Induced neonatal seizures diminish silent synapses and long-term potentiation in hippocampal CA1 neurons. Journal of Neuroscience, <\/em>31(50): 18211-18222.<\/p>\n\n\n\n

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