Dr. Hongyu Sun received his MD from Tianjin University in 1997 and his PhD in Neuroscience from Carleton University in 2008. He completed his postdoctoral training at Boston Children's Hospital / Harvard Medical School. He later joined the Carleton Neuroscience department in 2016.
Eligible to supervise at the undergraduate and graduate level.
The Sun Lab focuses on understanding how early-life experiences affect brain development and cause long-term psychiatric and neurological consequences. They use a combination of electrophysiological, behavioral, neuroimaging, modeling, and molecular biology techniques to address these questions.
Sun H, Takesian A, Wang TT, Lippman-Bell JJ, Hensch TK, Jensen FE. (2018) Early seizures prematurely unsilence auditory synapses to disrupt thalamocortical critical period plasticity. Cell Report. 23(9):2533-2540.
Sun H, Juul H, Jensen FE. (2016) Models of hypoxia and ischemia-induced seizures. Journal of Neuroscience Method, 15;260:252-60.
Sun H, Kosaras B, Klein PM, Jensen FE. (2013). Mammalian target of rapamycin complex 1 activation negatively regulates Polo-like kinase 2-mediated homeostatic compensation following neonatal seizures. PNAS, 110(13): 5199-204.
Talos DM, Sun H, Zhou X*, Fitzgerald E, Jackson M, Lan V, Joseph A, Jensen FE. (2012) Inhibition of the mammalian target of rapamycin (mTORC1) pathway prevents later life epilepsy and autism-like behavior following early life seizures. PLoS ONE, 7(5): e35885. doi:10.1371/journal.pone.0035885.
Zhou C, Lippman Bell JJ, Sun H*, Jensen FE. (2011) Hypoxia-Induced neonatal seizures diminish silent synapses and long-term potentiation in hippocampal CA1 neurons. Journal of Neuroscience, 31(50): 18211-18222.
